Pmr

March 21, 2018 | Author: scribmed | Category: Health Sciences, Wellness, Diseases And Disorders, Clinical Medicine, Medical Specialties


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Polymyalgia Rheumaticapitfalls in diagnosis PMR & GCA  Closely linked conditions  ? Different phases of the same disease PMR GCA  there is In situ production of cytokines in the temporal arteries of patients with PMR who do not have histological evidence of arteritis This suggests the presence of a subclinical vasculitis in PMR  . Clinical features of PMR  Systemic: Fever Anorexia Malaise Weight loss May present as PUO .  Proximal: Stiffness & pain in Shoulders  Neck  Hips  Worse with activity Difficulty getting out of bed .  Distal: Non-erosive arthritis (wrists & knees) CTS RS3PE . nodular.Clinical features of GCA         Headache: temporal / occipital Scalp tenderness ? Thickened. tender TA ? / absent pulse Jaw claudication: internal maxillary artery Tingling tongue: lingual artery Amaurosis fugax Unilateral permanent loss of vision other eye affected within 1-2 wks (ophthalmic & posterior ciliary arteries) . o  TA Bx  .phos.q -anaemia of ´chronic diseaseµ  Alk.Diagnosis: ESR o /CRPo .normal in 10%  Hb. n Fragmentation of internal elastic lamina . o Inflammatory infiltrate . o giant cell . Diagnostic criteria of PMR Bird et. al: Age > 65  ESR > 40  Onset within 2 weeks  Bilateral shoulder pain &/or stiffness  Morning stiffness > 1 hour  Bilateral upper arm tenderness  Depression &/or Wt loss  .  Diagnosis requires Any 3 of 7: Sensitivity 92% Specificity 80% . Problems in applying diagnostic criteria 40% have symptoms>3 months before referral 20% delay in hospital diagnosis>1 month 10% PMR with normal ESR    . Sensitivity 92% FN = 1 ² sensitivity = 8% Specificity 80% FP = 1 ² specificity = 20% . conditions frequently confused with polymyalgia? . Shoulder pain & stiffness Frozen shoulders  Rheumatoid arthritis  Fibromyalgia  Myxoedema  . Proximal weakness & tiredness Polymyositis  Thyrotoxic myopathy  Osteomalacia  Carcinomatous myopathy  . Raised ESR Myeloma  Malignancy  . followed up between 1995-1997 Haugeberg et.al .Prospective study of 50 patients in Norway with PMR symptoms. PMR TA collaginosis coxarthosis Shoulder tendinitis Prostate ca Liver mets unknown 1° myelodysplasia Lymphoma Total 40 2 1 1 1 1 1 2 1 50 Haugeberg.et. al . The observed frequency of malignancy in these patients was compared with the frequency of expected malignancy in the Norwegian population over the same period adjusted for age and sex. given by the national cancer registry . 6%  Frequency in study population = 10%  A two-tailed Fisher·s exact test: P = 0. Age & sex-adjusted frequency of malignancy in general population = 1.0013 . et. al .PMR TA collaginosis coxarthosis Shoulder tendinitis Prostate ca Liver mets unknown 1° myelodysplasia Lymphoma Total 40 2 1 1 1 1 1 2 1 50 Haugeberg. TA) = 8 = 16%  Specificity = 80%  FP = 20%  .Total non (PMR. A tricky disease .PMR. May present as PUO  May have normal ESR  May mimic hidden malignancy  EORA may present as PMR: Anti-CCP may differentiate  GCA May present with different arterial syndromes  . Features raising suspicion of other diagnoses: S Siebert et. al  younger  normal presenting age(< 65) inflammatory markers to low dose steroids raised ALP  non-response  persistently . Ca. if it does not investigate further  . serum electrophoresis and dipstick urinalysis ALP should to normal after 3/52 treatment. CK. ESR. U&Es. LFTs TFT. Patients with suspected PMR should have baseline: FBC. CRP.  Failure to respond to steroids should raise doubts about the diagnosis of PMR A good response to steroids does not necessarily confirm the diagnosis The presence of any atypical features should prompt more detailed investigation   .
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